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Nusbaum Inc transmitter contents
Figure 3. Proctolin directly excites MCN1 and CPN2. A, In normal saline MCN1 and CPN2 were silent. Super- fusion of proctolin (10 25 M) to the CoGs caused a depolarization of the membrane potential of these neurons and elicited rhythmic bursts of action potentials in them. MCN1 and CPN2 recordings are from different prepara- tions. B, With <t>transmitter</t> release sup- pressed in the CoGs because of the superfusion of low Ca 21 saline, MCN1 and CPN2 were weakly active. Super- fusion of proctolin (10 25 M) in this saline to the CoGs caused a depolar- ization of the MCN1 and CPN2 mem- brane potential and increased their fir- ing frequency. A and B are from different preparations, as are the MCN1 and CPN2 recordings.
Transmitter Contents, supplied by Nusbaum Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 86 stars, based on 1 article reviews
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1) Product Images from "Distinct Functions for Cotransmitters Mediating Motor Pattern Selection"

Article Title: Distinct Functions for Cotransmitters Mediating Motor Pattern Selection

Journal: The Journal of Neuroscience

doi: 10.1523/jneurosci.19-16-06774.1999

Figure 3. Proctolin directly excites MCN1 and CPN2. A, In normal saline MCN1 and CPN2 were silent. Super- fusion of proctolin (10 25 M) to the CoGs caused a depolarization of the membrane potential of these neurons and elicited rhythmic bursts of action potentials in them. MCN1 and CPN2 recordings are from different prepara- tions. B, With transmitter release sup- pressed in the CoGs because of the superfusion of low Ca 21 saline, MCN1 and CPN2 were weakly active. Super- fusion of proctolin (10 25 M) in this saline to the CoGs caused a depolar- ization of the MCN1 and CPN2 mem- brane potential and increased their fir- ing frequency. A and B are from different preparations, as are the MCN1 and CPN2 recordings.
Figure Legend Snippet: Figure 3. Proctolin directly excites MCN1 and CPN2. A, In normal saline MCN1 and CPN2 were silent. Super- fusion of proctolin (10 25 M) to the CoGs caused a depolarization of the membrane potential of these neurons and elicited rhythmic bursts of action potentials in them. MCN1 and CPN2 recordings are from different prepara- tions. B, With transmitter release sup- pressed in the CoGs because of the superfusion of low Ca 21 saline, MCN1 and CPN2 were weakly active. Super- fusion of proctolin (10 25 M) in this saline to the CoGs caused a depolar- ization of the MCN1 and CPN2 mem- brane potential and increased their fir- ing frequency. A and B are from different preparations, as are the MCN1 and CPN2 recordings.

Techniques Used: Saline, Membrane



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Nusbaum Inc transmitter contents
Figure 3. Proctolin directly excites MCN1 and CPN2. A, In normal saline MCN1 and CPN2 were silent. Super- fusion of proctolin (10 25 M) to the CoGs caused a depolarization of the membrane potential of these neurons and elicited rhythmic bursts of action potentials in them. MCN1 and CPN2 recordings are from different prepara- tions. B, With <t>transmitter</t> release sup- pressed in the CoGs because of the superfusion of low Ca 21 saline, MCN1 and CPN2 were weakly active. Super- fusion of proctolin (10 25 M) in this saline to the CoGs caused a depolar- ization of the MCN1 and CPN2 mem- brane potential and increased their fir- ing frequency. A and B are from different preparations, as are the MCN1 and CPN2 recordings.
Transmitter Contents, supplied by Nusbaum Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/transmitter+contents/10__1523_slash_jneurosci__19___16___06774__1999-29-21-29?v=Nusbaum+Inc
Average 86 stars, based on 1 article reviews
transmitter contents - by Bioz Stars, 2026-07
86/100 stars
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Figure 3. Proctolin directly excites MCN1 and CPN2. A, In normal saline MCN1 and CPN2 were silent. Super- fusion of proctolin (10 25 M) to the CoGs caused a depolarization of the membrane potential of these neurons and elicited rhythmic bursts of action potentials in them. MCN1 and CPN2 recordings are from different prepara- tions. B, With transmitter release sup- pressed in the CoGs because of the superfusion of low Ca 21 saline, MCN1 and CPN2 were weakly active. Super- fusion of proctolin (10 25 M) in this saline to the CoGs caused a depolar- ization of the MCN1 and CPN2 mem- brane potential and increased their fir- ing frequency. A and B are from different preparations, as are the MCN1 and CPN2 recordings.

Journal: The Journal of Neuroscience

Article Title: Distinct Functions for Cotransmitters Mediating Motor Pattern Selection

doi: 10.1523/jneurosci.19-16-06774.1999

Figure Lengend Snippet: Figure 3. Proctolin directly excites MCN1 and CPN2. A, In normal saline MCN1 and CPN2 were silent. Super- fusion of proctolin (10 25 M) to the CoGs caused a depolarization of the membrane potential of these neurons and elicited rhythmic bursts of action potentials in them. MCN1 and CPN2 recordings are from different prepara- tions. B, With transmitter release sup- pressed in the CoGs because of the superfusion of low Ca 21 saline, MCN1 and CPN2 were weakly active. Super- fusion of proctolin (10 25 M) in this saline to the CoGs caused a depolar- ization of the MCN1 and CPN2 mem- brane potential and increased their fir- ing frequency. A and B are from different preparations, as are the MCN1 and CPN2 recordings.

Article Snippet: The influence of several projection neurons on the rhythmically active neuronal network in the crab STG has been characterized, and their transmitter contents have been determined at least partly (Nusbaum and Marder, 1989a,b; Coleman and Nusbaum, 1994; Norris et al., 1994, 1996; Bartos and Nusbaum, 1997; Blitz and Nusbaum, 1997a; Blitz et al., 1999).

Techniques: Saline, Membrane